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Delayed wound healing by the absence of CD19 and the effect of growth factors

  • Yohei Iwata
  • , Ayumi Yoshizaki
  • , Kazuhiro Komura
  • , Fumihide Ogawa
  • , Kazuhiro Shimizu
  • , Shinichi Sato

Research output: Contribution to journalArticlepeer-review

Abstract

During the wound healing process, inflammatory cell infiltration promotes the wound healing by producing various cytokines and growth factors. Wound healing is delayed in mice lacking CD19, positive response regulator of B cells. This delayed wound healing was normalized by treatment with basic fibroblast growth factor. This indicates that basic fibroblast growth factor plays an important and critical role in the wound healing process.

Original languageEnglish
Pages (from-to)52-57
Number of pages6
JournalSkin Research
Volume8
Issue numberSUPPL. 11
Publication statusPublished - 02-2009
Externally publishedYes

UN SDGs

This output contributes to the following UN Sustainable Development Goals (SDGs)

  1. SDG 3 - Good Health and Well-being
    SDG 3 Good Health and Well-being

All Science Journal Classification (ASJC) codes

  • Dermatology
  • Infectious Diseases

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