SNX25 regulates proinflammatory cytokine expression via the NF-?B signal in macrophages

  • Kazuya Nishimura
  • , Tatsuhide Tanaka
  • , Shoko Takemura
  • , Kouko Tatsumi
  • , Akio Wanaka

Research output: Contribution to journalArticlepeer-review

11 Citations (Scopus)

Abstract

Innate immunity is the first line of defense against bacterial infection and is initiated by macrophages. Sorting nexin 25 (SNX25) is an SNX family member and is reported to negatively regulate TGF-ß signaling by enhancing TGF receptor degradation. However, few studies have focused on the relationship between SNX25 and the immune system. We knocked down SNX25 expression in macrophages and examined inflammatory cytokine expression, a hallmark of innate immunity, after lipopolysaccharide stimulation. SNX25 knockdown increased proinflammatory cytokine expression in RAW 264.7 cells. In addition, SNX25 knockdown activated the NF-?B signal by promoting ubiquitination of I?Ba. These results suggest that SNX25 inhibits the NF-?B signal and thereby regulates proinflammatory cytokine expression in macrophages.

Original languageEnglish
Article numbere0247840
JournalPloS one
Volume16
Issue number3 March
DOIs
Publication statusPublished - 03-2021
Externally publishedYes

All Science Journal Classification (ASJC) codes

  • General

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