TY - JOUR
T1 - Autoinflammatory diseases in dermatology
T2 - DITRA and CAMPS
AU - Sugiura, Kazumitsu
N1 - Publisher Copyright:
© 2017 The Japan Society for Clinical Immunology.
PY - 2017
Y1 - 2017
N2 - Deficiency of interleukin thirty-six receptor antagonist (DITRA) and CARD14 mediated psoriasis (CAMPS) are autoinflammatory diseases in dermatology. The causative genes of DITRA and CMAPS have been identified recently. In this paper, IL36RN and CARD14, the causative gene for DITRA and CAMPS, respectively were explained. In addition, clinical features and therapies for generalized pustular psoriasis not associated with psoriasis vulgaris (GPP without PsV), and pityriasis rubra pilaris type V (PRP type V) were described. GPP without PsV and PRP type V are representative diseases of DITRA and CAMPS, respectively.
AB - Deficiency of interleukin thirty-six receptor antagonist (DITRA) and CARD14 mediated psoriasis (CAMPS) are autoinflammatory diseases in dermatology. The causative genes of DITRA and CMAPS have been identified recently. In this paper, IL36RN and CARD14, the causative gene for DITRA and CAMPS, respectively were explained. In addition, clinical features and therapies for generalized pustular psoriasis not associated with psoriasis vulgaris (GPP without PsV), and pityriasis rubra pilaris type V (PRP type V) were described. GPP without PsV and PRP type V are representative diseases of DITRA and CAMPS, respectively.
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U2 - 10.2177/jsci.40.169
DO - 10.2177/jsci.40.169
M3 - Article
C2 - 28747603
AN - SCOPUS:85026429888
SN - 0911-4300
VL - 40
SP - 169
EP - 173
JO - Japanese Journal of Clinical Immunology
JF - Japanese Journal of Clinical Immunology
IS - 3
ER -