Rac1 and Cdc42 capture microtubules through IQGAP1 and CLIP-170

Masaki Fukata, Takashi Watanabe, Jun Noritake, Masato Nakagawa, Masaki Yamaga, Shinya Kuroda, Yoshiharu Matsuura, Akihiro Iwamatsu, Franck Perez, Kozo Kaibuchi

研究成果: ジャーナルへの寄稿学術論文査読

512 被引用数 (Scopus)

抄録

Linkage of microtubules to special cortical regions is essential for cell polarization. CLIP-170 binds to the growing ends of microtubules and plays pivotal roles in orientation. We have found that IQGAP1, an effector of Rac1 and Cdc42, interacts with CLIP-170. In Vero fibroblasts, IQGAP1 localizes at the polarized leading edge. Expression of carboxy-terminal fragment of IQGAP1, which includes the CLIP-170 binding region, delocalizes GFP-CLIP-170 from the tips of microtubules and alters the microtubule array. Activated Rac1/Cdc42, IQGAP1, and CLIP-170 form a tripartite complex. Furthermore, expression of an IQGAP1 mutant defective in Rac1/Cdc42 binding induces multiple leading edges. These results indicate that Rac1/Cdc42 marks special cortical spots where the IQGAP1 and CLIP-170 complex is targeted, leading to a polarized microtubule array and cell polarization.

本文言語英語
ページ(範囲)873-885
ページ数13
ジャーナルCell
109
7
DOI
出版ステータス出版済み - 28-06-2002
外部発表はい

All Science Journal Classification (ASJC) codes

  • 生化学、遺伝学、分子生物学一般

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